Jim Chinnis said:Well...the evidence *supports* causality, but is not
conclusive by a long shot. The causality argument is
*supported* by the fact that we understand how the
vitamins drop homocysteine. So when the vitamins are given
to hyperhomocysteinemic individuals and cIMT falls, we
know that the associated homocysteine drop wasn't caused
by the cIMT drop in some way... But we still don't know
that the cIMT drop was due to the homocysteine drop. The
study seems to me to rule out causality in the direction
of cIMT affects homocysteine, so it strengthens support to
causality in the other direction.But only support or suggestion. No proof.
--
Jim Chinnis Warrenton, Virginia, USA
Is it possible then with this interpretation that
atherosclerosis itself may cause elevated homocysteine? If
so, then the vitamins may be working through a completely
different pathway than homocysteine metabolism, correct?
--
Winning against heart attack and stroke
sonoscore.comsonoscore.comOpen ↗
Dr. Blanchard said, in response to Phantom Jim "Is it possible then
with this interpretation that atherosclerosis itself may cause
elevated homocysteine?"
Dr. Blanchard this was my understanding.
B'adant