General fitness, health and nutrition · Public discussion

Animal model of diabetes

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6 January 2005
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  1. Rinsho Ketsueki. 1989 Aug;30(8):1115-27. Related Articles, Links

    [Pathogenesis and mechanism of iron overload: ferric nitrilotriacetate,
    hemosiderin, active oxygen, and carcinogenesis]

    [Article in Japanese]

    Awai M.

    Iron overload is found clinically in such conditions as hemochromatosis
    and sideroblastic anemia, and after long term repeated transfusion in
    aplastic anemia. An animal model of iron overload was successfully
    developed in rats and rabbits by repeated intraperitoneal injections of
    ferric nitrilotriacetate (Fe3+-NTA). This procedure induced a diabetic
    state with hyperglycemia, ketonemia, glycosuria and ketonuria. Blood
    venesection on these rats reduced the iron load in the liver and
    pancreas, and ameliorated the general diabetic symptoms. A single
    injection of Fe3+-NTA in rats induced a temporary elevation in plasma
    iron concentration, lipid peroxidation in the perfused liver homogenate
    expressed by malondialdehyde (MDA) formation, blood GOT, GPT, ALP and
    gamma-GTP sequentially. Fe3+-NTA uptake in the liver caused membrane
    lipid peroxidation, and subsequently produced a transit liberation of
    liver cell enzymes, although the incorporated liver Fe3+-NTA was only
    1% of the injected dosage (7.5 mg iron/kg BW) at 3 hr after injection.
    The direct toxic effect of Fe3+-NTA to living cells was examined using
    cultured normal rat liver parenchymal cells (RL-34). Marked cytolysis
    was found in cells exposed to more than 25 micrograms of iron through
    Fe3+-NTA/ml. At 50 micrograms iron of Fe3+-NTA/ml, most cells were
    lethally injured and the remaining cells were piled up and aggregated
    at 15 days. They grew on soft agar culture, and when inoculated
    subcutaneously to five newly born rats a subcutaneous tumor developed
    in all animals within three weeks. Lung metastases were found in three
    of five inoculated rats. A spin trapping technique with electron spin
    resonance (ESR) on Fe3+-NTA employing 5, 5-dimethyl-l-pyrroline-N-oxide
    (DMPO) yielded a spin adduct with three doublets (DMPO-Z) which
    corresponded to singlet oxygen. By ESR in the presence of H2O2, the
    Fe3+-NTA solution strongly generated hydroxyl radical. The production
    of active oxygen species by Fe3+-NTA solution may explain the toxicity
    and carcinogenicity of Fe3+-NTA. The majority of stainable iron in the
    iron overloaded tissue was hemosiderin (Hs). We tried to purify the Hs
    from multi-transfused human spleen by the method of Weir et al. The
    purified Hs did not show a DMPO-OH adducts in the presence of H2O2 and
    DMPO on ESR measurement. The Hs iron was solubilized with several
    biological ligands in an acidic state in the presence of a reducing
    reagent like glutathione. Solubilized Hs iron produced iron chelate
    complexes which resulted in OH radicals production in the presence of
    H2O2 in acidic conditions below pH 5.5.(ABSTRACT TRUNCATED AT 400
    WORDS)

    Publication Types:
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    Review, Tutorial

    PMID: 2689676 [PubMed - indexed for MEDLINE]

    --------------------------------------------------------------------------------
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    Tom

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