Human model of MS ?

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Subject: Re: Human model of MS ?
From: Rich Andrews [email hidden]
Date: 9/29/2003 6:13 PM Mountain Daylight Time
Message-id: <[email hidden]>

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Med Hypotheses. 1984 Nov;15(3):293-9. Related Articles, Links

The aetiology of SMON may involve an interaction between clioquinol and
environmental metals.

Tjalve H.

Clioquinol can increase the penetration of metallic cations through cellular
membranes by forming lipophilic metal-chelates. It is suggested that the
pathogenesis of SMON may involve an accumulation of injurious metals in nervous
tissues due to facilitated uptake by complex formation with clioquinol. The
Japanese have been more heavily exposed to metals by environmental pollution
than the inhabitants of other countries, which may explain the prevalent
occurrence of SMON in Japan.

PMID: 6240588 [PubMed - indexed for MEDLINE]

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