Mirek Fidler said:Quoted message said:consisted of a plant-based diet, low in salt and fat, with
monounsaturates as the principal fat." PMID: 11710359
Well, in the above list I do not see any significant source of
monounsatured fat. So once again, information seems incosistent.
They use a combination of low erucic rapeseed and soy oil for cooking.
There is some inconsistency--some values refer only to the diets
of the elders (at 26% dietary fat), while some refer to the population
as a whole. And of course now, the data are not very applicable,since
they are eating at McDonald's and have lost their edge.
But, they are not an isolated case. There are centenarian
populations with even lower total fat and meat intakes.
Chin Med J (Engl). 2001 Oct;114(10):1095-7.
A survey of the dietary nutritional composition of centenarians.
Chen C
PMID 11677774
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=11677774
The full paper is here--
http://www.cmj.org/information/full.asp?pmid=2001101095
Check out table 1:
food kcal
dried sweet potato slices 714
cereals 209
beans 118
meats 27
fat 135
vegetables 116
total 1419
total protein grams 39 (11%)
total fat grams 29.2 (19%)
total carbohydrate grams 227.6 (70%)
Also consider the work of Blankenhorn:
Blankenhorn DH, Johnson RL, Mack WJ, el Zein HA, Vailas LI. The
influence of diet on the appearance of new lesions in human coronary
arteries.
JAMA. 1990 Mar 23-30;263(12):1646-52.
PMID: 2407875 [PubMed - indexed for MEDLINE]
This study used angiograms to measure development of atherosclerotic
lesions along with dietary recall. This is a more stringent measure
than a change in serum lipid profiles.
"Each quartile of increased consumption of total fat and polyunsaturated
fat was associated with a significant increase in risk of new lesions.
Increased intake of lauric, oleic, and linoleic acids significantly
increased risk. Subjects in the Cholesterol Lowering Atherosclerosis
Study in whom new lesions did not develop increased dietary protein to
compensate for reduced intake of fat by substituting low-fat meats and
dairy products for high-fat meats and dairy products."
"These results indicate that when total and saturated fat intakes are
reduced to levels recommended by the National Cholesterol Education
Program, protein and carbohydrate are preferred substitutes for fat
calories, rather than monounsaturated or polyunsaturated fat."
The actual data from this paper are as follows:
diet intake associations w/ new lesion development
OR 95% confidence interval
total kcal 1.0 1.0-1.0
calories from protein, % 0.72 .57-.92
calories from carbohydrate % 0.98 .92-1.04
calories from total fat % 1.11 1.02-1.2
calories from saturated fat % 1.13 0.95-1.35
calories from PUFA % 1.28 1.07-1.53
calories from MUFA % 1.15 0.97-1.38
calories from alcohol % 0.97 0.87-1.08
cholesterol, mg/day 1.0 0.99-1.0
calcium, mg/day 1.0 1.0-1.0
The paper does not provide details about the diet,
only that the PUFA fraction was mostly from refined
oils high in omega6. But, the results for MUFA are
not good.
Hans Dehmelt has stated:
"Humans should ask themselves how wise it is to deviate widely from a
diet on which the primate line has flourished for many tens of millions
of years *and produced them*."
The diet of a wild primate line with GI tracts very similar
to ours, the howler monkey, is approximately 14% protein,
22.5% fat (comprised of about 30% palmitic, 23% linoleic,
16% alpha linolenic, 15% oleic and 16% other fatty acids),
and largely plant based, lightly supplemented by small mammals,
nuts, eggs, and termites.
Man has displayed the ability to thrive on vastly different
diets. However, it might perhaps be best not to deviate too
much from this base.
The diets of the Okinawan Elders and the Chinese centenarians
in the Chen study are close to this howler monkey base. I know
of zero studied centenarian populations that stray far
from this base.
A total fat intake in the low 20s% is likely to be characteristic
of the paleolithic people as well:
http://www.ajcn.org/cgi/content/full/70/3/560S
See figure 1.
Suddenly we are supposed increase our monounsaturated fat
intakes to very high levels in order to achieve optimal
health? I think not. Rather, it seems more likely that
elevated MUFA may be a better choice, in the grand
scheme of things, than elevated refined carbohydrate
or elevated saturated or trans fat intakes.
But, increased intake of unrefined carbohydrate sources,
more preferrably from vegetables than domesticated fruits
or whole grains, in the context of a diet adequate in protein,
possibly with more vegetable than animal protein, rich in soluble
fibers, and adequate, but not elevated, in saturated fats, MUFA,
and PUFA, is likely to be the best choice of all.
(Jenkins et al, PMID 11288049)